I. Introduction
Ventricular (VT) and atrial tachycardias (AT) are some of the most common clinical cardiac tachyarrhythmias. They are characterized by abnormal and fast muscular activity, with an organized electrical activity resulting from altered electrical propagation. Two dominant mechanisms have been identified to drive arrhythmias [1], [2]: (i) reentrant activation waves that rotate around a functional or anatomical obstacles (eg. an area of scar or a valve); and (ii) abnormal autonomous focal discharges of a group of cells that triggers abnormal activation wavefronts. As these mechanisms have specific propagation patterns, the mapping of these patterns could be a relevant tool to identify arrhythmic mechanisms, and localize pathological tissues for catheter ablation or surgical treatments.